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  • Allergic Rhinitis2
  • Chronic Rhinosinusitis8
  • Acoustic Neuroma (Vestibular Schwannoma)18
  • Acute Otitis Media35
  • Anatomy and Physiology of Hearing43
  • Assessment of Balance10
  • Audiology and Hearing Investigations11
  • Auditory Neuropathy Spectrum Disorder and Auditory Processing Disorder42
  • Autoimmune Inner Ear Disease (AIED)4
  • Benign Paroxysmal Positional Vertigo (BPPV)8
  • Bone-Anchored Hearing Aids (BAHA)8
  • Cholesteatoma12
  • Cochlear Implantation in Children11
  • Cochlear implantation12
  • Eustachian Tube Dysfunction (ETD)4
  • Hearing Aids and Auditory Rehabilitation8
  • Mastoiditis and Complications of Otitis Media4
  • Middle Ear Implants
  • Ménière's Disease10
  • Necrotising Otitis Externa3
  • Non-Organic Hearing Loss1
  • Ossicular Chain Pathology and Reconstruction
  • Otitis Externa1
  • Otitis Media with Effusion (Glue Ear)10
  • Otosclerosis13
  • Sudden Sensorineural Hearing Loss (SSNHL)12
  • Temporal Bone Fractures
  • Tinnitus and Hyperacusis1
  • Tympanoplasty and Mastoidectomy
  • Aetiology of Head and Neck Cancer27
  • Cervical Lymphadenopathy1
  • Adenoidal Hypertrophy and Adenoidectomy24
  • Age-Appropriate Paediatric Hearing Assessment16
  • Cleft Palate (ENT Considerations)8
  • Neonatal and Infant Airway Disorders
  • Paediatric Tracheostomy and Airway Management8
  • Tonsillitis and Tonsillectomy
Notes
HomeNotesBenign Paroxysmal Positional Vertigo (BPPV)

Otology

Benign Paroxysmal Positional Vertigo (BPPV)

Updated 6 September 2026

Summary

  • Brief vertigo triggered by lying back, rolling in bed or looking up suggests benign paroxysmal positional vertigo (BPPV). Confirm the affected canal with positioning tests and offer a matching repositioning manoeuvre. AAO-HNS 2017

  • Hearing loss, focal neurology or persistent spontaneous vertigo require a separate explanation, even when BPPV coexists. See Imaging indications.

  • Posterior canal disease is commonest; horizontal, anterior, bilateral and multicanal disease also occur.

    More detail

    Canalolithiasis produces abnormal endolymph movement when gravity moves the particles. Cupulolithiasis can cause more sustained positional nystagmus.

1. History

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